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            目錄:北京索萊寶科技有限公司>>抗體>>Polyclonal Antibody>> K101528PAnti-HS71A Polyclonal Antibody

            Anti-HS71A Polyclonal Antibody
            • Anti-HS71A Polyclonal Antibody
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            • SOLARBIO/索萊寶 品牌
            • K101528P 型號
            • 生產(chǎn)商 廠商性質(zhì)
            • 北京市 所在地
            規(guī)格

            屬性

            供貨周期:現(xiàn)貨 規(guī)格:50ul/100ul 貨號:K101528P 應(yīng)用領(lǐng)域:醫(yī)療衛(wèi)生,環(huán)保,化工,生物產(chǎn)業(yè),農(nóng)林牧漁 主要用途:WB

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            更新時間:2022-08-15 09:20:40瀏覽次數(shù):883評價

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            供貨周期 現(xiàn)貨 規(guī)格 50ul/100ul
            貨號 K101528P 應(yīng)用領(lǐng)域 醫(yī)療衛(wèi)生,環(huán)保,化工,生物產(chǎn)業(yè),農(nóng)林牧漁
            主要用途 WB
            Anti-HS71A Polyclonal Antibody
            應(yīng)用 WB
            稀釋比例WB 1:2500.
            交叉反應(yīng)Human

            Anti-HS71A Polyclonal Antibody

            英文名稱Anti-HS71A Polyclonal Antibody
            宿主Rabbit
            別名ABL;JTK7;bcr/abl;c-ABL;c-ABL1;p150;v-abl
            應(yīng)用WB
            稀釋比例WB 1:2500.
            交叉反應(yīng)Human
            蛋白分子量70kDa
            Gene ID3303/3304.
            保存Store at -20°C. Avoid freeze / thaw cycles.
            儲存液Buffer: PBS with 0.03% Proclin300, 50% glycerol, pH7.3.
            純化方法Affinity purification
            亞型IgG
            免疫原A synthetic peptide of human HS71A
            性狀液體
            Public Immunogen RangeA synthetic peptide of human HS71A
            Subcellular LocationsCytoskeleton Nucleus
            Swiss ProtP0DMV8
            克隆類型Polyclonal Antibody
            背景資料In cooperation with other chaperones, Hsp70s stabilize preexistent proteins against aggregation and mediate the folding of newly translated polypeptides in the cytosol as well as within organelles. These chaperones participate in all these processes through their ability to recognize nonnative conformations of other proteins. They bind extended peptide segments with a net hydrophobic character exposed by polypeptides during translation and membrane translocation, or following stress-induced damage. Essential for STUB1-mediated ubiquitination and degradation of FOXP3 in regulatory T-cells (Treg) during inflammation. Negatively regulates heat shock-induced HSF1 transcriptional activity during the attenuation and recovery phase period of the heat shock response.

             

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