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乙型流感病毒IgG酶聯(lián)免疫法檢測(cè)試劑盒
廣州健侖生物科技有限公司
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乙型流感病毒IgG酶聯(lián)免疫法檢測(cè)試劑盒
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乙型流感病毒IgG酶聯(lián)免疫法檢測(cè)試劑盒
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【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】 歐
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【騰訊 】
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103室
瞳孔 形狀、大小、邊緣、光反應(yīng)(直接、間接、輻輳)。瞳孔區(qū)有無滲出物、色素等。
7.晶體 是否存在，位置及透明度。
在暗室內(nèi)用直接或間接檢眼鏡進(jìn)行檢查。觀察玻璃體有無混濁、出血、液化、變性、異物、寄生蟲等。檢查眼底應(yīng)注意視盤、視網(wǎng)膜血管及黃斑及眼底全貌，有無炎癥、出血、滲出、變性、畸形等。
(四)特殊檢查
1.裂隙燈顯微鏡檢查 能進(jìn)一步觀察眼部各組織的微細(xì)變化。注意角膜、房水、晶體及前部玻璃體的微細(xì)病變。結(jié)合角膜染色法(2%熒光素溶液染色)，以鑒別角膜新鮮與陳舊病變。結(jié)合前房角鏡觀察前房角的變化，結(jié)合三面鏡觀察眼底各部位的改變。
2.視野 包括中心視野和周邊視野，以了解視神經(jīng)、視網(wǎng)膜及視路的功能變化。
3.檢影、試鏡 了解屈光狀態(tài)。
4.眼壓及眼球突出度的測(cè)量 青光眼的眼壓測(cè)量是*的，但對(duì)診斷難以明確者，尚需進(jìn)一步做24 h晝夜眼壓曲線、房水流出易度C值測(cè)定以及眼壓激發(fā)試驗(yàn)。
為了明確診斷或追究病因，血壓、血、尿常規(guī)、紅細(xì)胞沉降率、血糖、結(jié)核菌素試驗(yàn)、甲狀腺功能、病理檢查等均有重要參考價(jià)值。
1.眼底熒光血管造影 能進(jìn)一步了解眼底血液循環(huán)(達(dá)毛細(xì)血管水平)的細(xì)微結(jié)構(gòu)、動(dòng)態(tài)變化以及功能上的改變，為眼底病提出更多、更詳盡的診斷依據(jù)。
2.視覺電生理檢查 包括視網(wǎng)膜電圖(ERG)、眼電圖(EOG)、視誘發(fā)電位(VEP)等，以了解視網(wǎng)膜及視路功能。
3.影像檢查 包括細(xì)菌、眼眶X線檢查，超聲探查以型超聲(A型超聲、B型超聲、超聲多普勒)，CT掃描，磁共振成像(MRI)等。可以顯示眼部結(jié)構(gòu)和病理變化，對(duì)眼部不透明組織可達(dá)到直接視診目的。視網(wǎng)膜變性是視網(wǎng)膜裂孔形成的基礎(chǔ)。視網(wǎng)膜的周邊部和黃斑部血液供應(yīng)較少，相對(duì)容易產(chǎn)生視網(wǎng)膜變性。下列視網(wǎng)膜變性是誘發(fā)視網(wǎng)膜脫離的危險(xiǎn)因素。（1）格子樣變性：格子樣變性與視網(wǎng)膜脫離關(guān)系z(mì)ui為密切。由此產(chǎn)生網(wǎng)膜裂孔者占孔源性脫離眼的40％，約7％的正常眼存在格子樣變性。多見于顳側(cè)或顳上象限的赤道部與鋸齒緣間，呈梭形或條狀，長(zhǎng)軸與鋸齒緣平行，病灶內(nèi)視網(wǎng)膜變薄，有許多白色線條，交錯(cuò)排列成網(wǎng)格狀。這些線條實(shí)際上是已經(jīng)閉塞或帶有白鞘的末梢血管。病灶內(nèi)有時(shí)可見色素團(tuán)，來源于視網(wǎng)膜色素上皮。
Pupil shape, size, edge, photoreaction (direct, indirect, convergence). Pupil area with or without exudates, pigments and so on.
7. Crystal existence, location and transparency.
In the dark room with direct or indirect ophthalmic examination. Observe vitreous with or without opacity, bleeding, liquefaction, degeneration, foreign bodies, parasites and so on. Check the fundus should pay attention to the optic disc, retinal blood vessels and macular and fundus, with or without inflammation, bleeding, exudation, degeneration, deformity and so on.
(D) special inspection
Slit lamp microscopy can further observe the fine changes in the organization of the eye. Attention to the cornea, aqueous humor, and the front of the lens vitreous micro-lesions. Combined with corneal staining (2% fluorescein staining) to identify fresh and old corneal lesions. Combined with the changes of anterior chamber angle under the angle goggles, combined with the three mirror to observe the changes of various parts of the fundus.
2. Field of vision including the center of vision and the surrounding field of vision, in order to understand the optic nerve, retinal and visual pathway changes in function.
3. retinoscopy, auditions to understand the refractive status.
4. IOP and protrusion of the glaucoma measurement of intraocular pressure measurement is essential, but the diagnosis is difficult to clear, still need to do 24 h day and night intraocular pressure curve, atrial outflow of ease C value determination and intraocular pressure Excite the test.
In order to confirm the diagnosis or investigate the cause, blood pressure, blood, urine routine, erythrocyte sedimentation rate, blood glucose, tuberculin test, thyroid function, pathological examination have important reference value.
1. Fundus fluorescein angiography can further understand the fundus blood circulation (up to the level of capillaries) the fine structure, dynamic changes and functional changes for the fundus put forward more more, more detailed diagnosis basis.
2. Visual electrophysiological examination including electroretinography (ERG), electro-oculography (EOG), evoked potential (VEP), etc., in order to understand the retina and visual pathway.
3. Imaging examination including bacteria, orbital X-ray examination, ultrasound to type ultrasound (A ultrasound, B ultrasound, ultrasound Doppler), CT scan, magnetic resonance imaging (MRI) and so on. Can show the ocular structure and pathological changes, ophthalmic ophthalmic organizations can achieve direct visual inspection purposes. Retinal degeneration is the basis of retinal breaks. Retinal peripheral and macular blood supply less prone to retinal degeneration. The following retinal degeneration is a risk factor for retinal detachment. (1) lattice degeneration: lattice degeneration and retinal detachment most closely. The resulting rupture of the omentum accounts for 40% of the holes removed from the eye, and about 7% of the normal eyes have the lattice-like degeneration. More common in the temporal or superior quadrant of the equator between the serrated edge, was spindle or strip, long axis and the serrated edge parallel to the thinning of the lesion within the retina, there are many white lines, staggered arranged in a grid. These lines are actually occluded or white sheathing peripheral blood vessels. Pigmented mass sometimes seen within the lesion, derived from the retinal pigment epithelium.